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It is largely because they act on similar biological pathways (appetite regulation, digestion)

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These molecules mimic the action of two natural hormones involved in metabolism and appetite regulation: GLP-1 (Glucagon-Like Peptide-1) and GIP (Glucose-Dependent Insulinotropic Polypeptide)

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GLP-1 receptor agonists have known satiety-promoting effects due to central action of the neuropeptide GLP-1 [1], with reduced activation of appetite- and reward-related brain areas in response to food [18]