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Background and Summary of Retrospective Studies Homocystinuria caused by congenital CBS and MTHFR defects or by genetic alteration of cobalamin metabolism is characterized by clinical problems including frequent arterial and venous thromboembolism and premature atherosclerosis (McCully, 1983

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MTs participate in the process of carcinogenesis and play critical roles in tumor growth, progression, metastasis, and drug resistance (Fig

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Early activation of peripheral monocytes with hallmarks of M1 and M2 monocytic cells in excessive alcohol drinkers: a pilot study