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Recent evidence suggests that cellular senescence plays a role in promoting fibrosis, especially in aged tissues, where stress-induced senescent myofibroblasts may exhibit delayed clearance with age

The intricate and not entirely understood interaction between the host and the microbiome poses the main limitation of these approaches

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Improving joint recovery and mobility is an area of BPC-157 research with notable preclinical support across tendon, ligament, and cartilage injury models

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