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Research in animal models suggests BPC-157 may promote fibroblast activity and increase the rate at which collagen is synthesised at injury sites
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doi: 10.1097/RCT.0000000000000406 53 SolomonJMarinDRoy ChoudhuryKPatelBSameiE
When it comes to anti-aging, BPC-157s cellular repair capabilities cant be ignored
Another proposed mechanism involves the binding of homocysteine to the collagenous matrix of bone, which may modify collagen properties and reduce bone strength (reviewed in 113)
