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The Harvard researchers speculated that the TMAO from the high dietary choline [intake] may increase inflammation, and this may promote progression of prostate cancer to lethal disease. In fact, just yesterday, in the New England Journal of Medicine, that same Cleveland Clinic research team that did the carnitine study repeated the study
[DOI] [PMC free article] [PubMed] [Google Scholar] 12.ODonnell-Luria AH, Lin AP, Merugumala SK, et al
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Au cours d'un repas
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2023ZD04073, G.J.), the National Key R&D Program of China (grant no
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Oleic acid recovery dose = 14 C-Oleic acid (20 L 1 mCi/mL) + KRP (80 L)
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In particular, AR signaling influences the fates of citrate that uniquely characterizes normal and malignant prostatic metabolism (i.e., mitochondrial export and extracellular secretion in normal prostate, mitochondrial retention and oxidation to support oxidative phenotype of primary PCa, and extra-mitochondrial interconversion into acetyl-CoA for fatty acid synthesis and epigenetics in the advanced PCa)
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