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The results, while modest, were consistent with the peptide's selective mechanism.[4] The peptide appeared especially useful in the study for mobilizing visceral and abdominal fat the type most resistant to traditional diet and exercise

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In animal studies, blocking NNMT raised NAD+ levels and led to lower fat mass without cutting food intake or lean muscle

Doubling the dose of MOTS-c wont resolve NAD+ depletion caused by NNMT overactivity, and increasing 5-Amino-1MQ wont activate AMPK if signaling pathways are suppressed