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Activation of FXR suppresses hepatic gluconeogenesis by downregulating key enzymes, including phosphoenolpyruvate carboxykinase and glucose- 6-phosphatase, thereby reducing blood glucose levels [73, 74]

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It was therefore concluded that NiCl 2 induces p38 MAPK-dependent lytic cell death and stimulates markers of premature eryptosis, which may explain the changes in hematological parameters observed in poisoning victims [420]

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