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1 In vitro studies conducted in Vero cells were excluded because of the inability for these cells to produce type I interferon responses, making them unsuitable for assessing the immunomodulatory effects of HCQ and CQ

Considering that many chronic age-related diseases appear to have some degree of mitochondrial dysfunction as part of their etiology, it is notable that alterations in mGSH homeostasis may be a central factor in this dysfunction

We reported GSH concentration within the different brain areas investigated for HC subjects found in the literature (Table 1)

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2019), localization (Krahmer et al