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In a previous study, oral administration of LA has been shown to simultaneously activate SIRT1 and AMPK to decrease lipids, and another study has shown that LA can target the liver sulfane sulfur/hydrogen sulfide signaling pathway to prevent liver damage caused by diabetes
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The last step in the production of TMAO can also be targeted, albeit with the known side effects of elevated TMA levels
Following the initial dehydrogenation, the resulting long-chain enoyl-CoAs undergo hydration, a second dehydrogenation step, and finally thiolytic cleavage catalyzed by mitochondrial trifunctional protein (MTP) [10]
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