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Atypical cobalamin deficiency: subtle biochemical evidence of deficiency is commonly demonstrable in patients without megaloblastic anemia and is often associated with protein-bound cobalamin malabsorption

What the generic summaries miss is the architecture of mechanism the specific receptor interactions, the molecular signaling cascades, the crosstalk between nitric oxide pathways and angiogenic growth factors that explains why a fifteen-amino-acid fragment produces effects observed across tissues as different as gastric mucosa, tendon, skeletal muscle, and peripheral nerve