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Nevertheless, hypoxia may have detrimental effects on the central nervous system and brain function 1, 5, 6

We show that the inflammatory response elicited by elevated intracellular lipids in skeletal muscle is repressed in Cpt1b m/ mice, strongly supporting the hypothesis that mitochondrial processing of fatty acids is essential for the lipid-induction of inflammation in muscle

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Given that impaired autophagy and dysregulated mucin secretion are key contributors to barrier dysfunction, TB-500s regenerative and anti-inflammatory actions may indirectly support mucin2 production and mucus layer integrity, though direct evidence in colitis models remains limited

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