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Under the current findings MLE studies provide evidence that a complex mixture acts within an hormetic fashion for multiple cell types for a range of endpoints
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During development and progression of atherosclerosis, Nrf2 signaling modulates many physiological and pathophysiological processes, like regulation of lipid homeostasis, CD36 gene expression regulation, foam cell formation, macrophage polarization, immunity regulation (Th2 differentiation and inhibition of pro-inflammatory gene expression through NFB down-regulation), redox regulation and inflammation, improvement of endothelial dysfunction, as well as GSH synthesis and utilization (234245)
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These results confirmed that Ang II effectively induced senescent phenotypes in primary cultured HAECs
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