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In addition, the expression levels of antiapoptotic Bcl-2 and caspase-8 decreased, whereas expression levels of BAK, BAX, BID, and caspase-3 were significantly increased in both A549 and L132 cells, suggesting gliotxin induce the apoptosis of A459 and L132 cells via the activation of caspases and the Bax pathway, also implicating mitochondrial damage [47]

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For decades, weve understood mitochondria primarily as energy producers

Exploiting mucosal surfaces for the development of mucosal vaccines

They can suggest alternative antibiotics or adjust dosages to prevent negative interactions
