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A later study elucidated that compounds CHIR, E616452, and forskolin firstly worked in a cooperative manner to activate Sox7
gallinarum in germ-free mice also recapitulated an LGS-like phenotype with enhanced bacterial translocation to the liver, presumably due to the induction of Hp/zonulin and the reciprocal downregulation of TJ-related molecules (e.g., Cldn3 and Ocln )
