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Moreover, Brusatol effectively managed NSCLC by augmenting the tricarboxylic acid cycle as well as Complex I activity within the mitochondrial electron transport chain, consequently enhancing the susceptibility of NSCLC cells to ferroptosis induced by cysteine deprivation

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1993c) and some of the GABA released by neurons is taken up and metabolized by astrocytes, maintenance of GABAergic neurotransmission also relies on anaplerosis (Schousboe et al

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[DOI] [PMC free article] [PubMed] [Google Scholar] 140.Granell R., Heron J., Lewis S., Davey Smith G., Sterne J.A.C., Henderson J
