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In conclusion, this is a rare though probably underdiagnosed cause of metabolic acidosis with increased anion gap

& Penfield, W

Nevertheless, beyond myelination, DA neuron interactions with OLs and OPCs in the SNpc have been suggested to have a prominent role in the etiology of PD

Brain ultrastructural changes during HE have been extensively studied, including edema of endothelial cells, changes in morphology of pericytes and perivascular astrocytes feet, with an increase number of vacuoles and vesicles in both cell types

Our study revealed that allicin altered lipid metabolism in the peri-infarct cortex and decreased arachidonic acid metabolism like PGE2, 5-HPETE, TXA2, and TXB2 (Fig

Sci China Life Sci (2020) 63(1):117