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Mutant proteins are reported to block the transport of nuclear-encoded mitochondrial proteins to mitochondria, interact with mitochondrial proteins and disrupt the electron transport chain, induce free radicals, cause mitochondrial dysfunction, and, ultimately, damage neurons

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Este desequilibrio podra ser precursor de enfermedades crnicas como diabetes, cncer y artritis reumatoides
Selenocysteine is present at the catalytic site of glutathione peroxidase, and selenium av

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