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The more permeable lipophilic ester prodrugs, POM-SF and POM-HEX, are converted intracellularly to the direct enolase inhibitors SF-2312 and HEX, respectively, and have substantially increased cellular potencies over their cognate phosphonates (Lin et al., 2020)

Exp Clin Cardiol 2011;16(3):87-91

Data collected from in vitro and in vivo tumor models suggest that these typically take place by means of three mechanisms: the occurrence of specific genetic lesions, including translocations, gene amplifications and activating mutations

[45] investigated similar parameters in nonobese women with a family history of type 2 diabetes and normal glucose values

Individual clinical appropriateness must be determined by a licensed provider based on personal health factors

Discussion Our findings are suggestive of sex-specific adverse programming of growth and metabolic milieu as early as fetal life and in the postnatal period, with exposure to prenatal T excess during early to mid-gestation