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While senescence acts as a protective mechanism against tumorigenesis, the accumulation of senescent cells and their inflammatory SASP can promote tumor progression.53 Combination therapies that target senescent cells in the tumor microenvironment and modulate the SASP could offer new therapeutic options in cancer treatment, potentially inhibiting tumor growth while enhancing the efficacy of existing therapies like chemotherapy and immunotherapy.194,217 Preclinical Evidence Supporting Combination Therapies In preclinical animal models, the combination of senolytic and senomorphic agents has shown promising results

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Nausea (most common, typically dose-dependent and transient) Diarrhea or constipation during dose escalation Vomiting, especially when starting or increasing dose Reduced appetite and early satiety Injection site reactions (redness, bruising) Fatigue, particularly in early weeks Rare: Pancreatitis discontinue immediately with severe abdominal pain Rare: Thyroid C-cell effects (contraindicated with MEN2 or thyroid cancer history) AOD-9604 is a modified C-terminal fragment (residues 176191, Tyr-hGH177-191) of human growth hormone, studied for lipolytic and anti-obesity properties

In vivo (PC-3 xenograft nude mice), garcinol decreased tumor volume and size and induced apoptosis [282]

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MiR-652-3p promotes bladder cancer migration and invasion by targeting KCNN3