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PHT induces atherosclerosis (AS) through the induction of endothelial dysfunction, and insulin resistance (IR)
Finally, we demonstrated that oral administration of 2-AB efficiently raises both circulating and myocardial glutathione levels and protects against doxorubicin-induced cardiomyopathy in mice
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On the contrary, GSTs exhibiting a cysteine as catalytic residue catalyze the opposite reaction (deglutathionylation) and exhibit thiol-transferase or reductase activities
