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Description
Overexpression of GPX4 will prevent BAY-induced cell death, while GPX4 knockdown potentiated cell death by increasing the accumulation of lipid products [112]

However, excess ROS produced under pathophysiological conditions damage proteins, lipids, and DNA

Mesenchymal stromal cell treatment attenuates repetitive mild traumatic brain injury-induced persistent cognitive deficits via suppressing ferroptosis

Structural Characteristics of High-Mobility Group Proteins HMGB1 and HMGB2 and Their Interaction with DNA

For instance, the introduction of different nanocarriers and the development of curcumin targeting mitochondrial derivatives (37,38)

This section discusses molecular mechanisms and therapeutic strategies for ovarian cancer, focusing on redox modification
