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Under conditions of high oxidative stress, BH4 is rapidly oxidized and destroyed, causing eNOS to "uncouple." When eNOS uncouples, it stops producing healthy nitric oxide and instead produces more destructive superoxide, accelerating vascular damage

Ubiquitination-Mediated Degradation of Sirt1 by Smurf2 Suppresses Crc Cell Proliferation and Tumorigenesis

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Taken together, various cell death pathways exist that in physiological conditions take part in the fine processes of development, differentiation, and removal of damaged or unfunctional cells and subcellular structures, or harmful microorganisms

Crit Rev Clin Lab Sci 38: 263355, 2001 [DOI] [PubMed] [Google Scholar] 103

Autoantibodies and Clinical Associations in Autoimmune Thyroid Diseases The pathophysiology and clinical presentation of autoimmune thyroid disorders are largely influenced by autoantibodies