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J Physiol Biochem (2019) 75(2):12533
In: (Eds), Special Issue on Zoonoses
TMAO activates the NLRP3 inflammasome through three distinct signaling mechanisms: mitochondrial dysfunction induced by Rac1-NOX2-dependent ROS production, which causes cytosolic release of oxidized mtDNA
The GIP receptor activation adds an additional metabolic dimension that appears to enhance fat oxidation and improve insulin sensitivity beyond what GLP-1 activation alone achieves
