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Here, we propose the misfolding of the mutant ATXN3 protein and the subsequent aggregation process, with eventual deposition of insoluble intracellular aggregates, to be the hub of the pathogenic process, and discuss how protein dyshomeostasis impacts key cellular pathways causing dysfunction and disease (Fig

We induced myocardial isoprenaline lesions (as described [24] with one or two applications of isoprenaline (75 mg/kg or 150 mg/kg s.c.))

doi: 10.1096/fba.2021-00022

Injectable peptides may provide consistent delivery and support systemic healing processes

Deficiency is more common than most people realize and is linked to persistent fatigue, brain fog, mood changes, and slow metabolism