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Azithromycin works by inhibiting bacterial protein synthesis (binding to the 50S ribosomal subunit in bacteria), which stops bacteria from growing and multiplying

It helps address symptoms such as

Euglycemic ketoacidosis has emerged as a concerning complication in this patient population, yet no study has directly compared ketoacidosis reporting signals between these two agents in non-diabetic patients using real-world pharmacovigilance data

Conclusion GLP-1 RAs may significantly lower the combined risk of mortality and hospitalization in patients with HFpEF