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Description
One of the most vexing issues in the field of inflammation research pertains to how the very bacteria and cells in the intestine that are required for host survival can also induce life-threatening inflammation
Why it Matters
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While it is well-established that intestinal GLP-1 cells can exert influence through paracrine mechanisms, recent evidence suggests the possible existence of synaptic-like connections between GLP-1 cells and peripheral neurons, including those of the vagus nerve
So before we move quickly to external GLP-1 support, a more important question is: Are we activating endogenous pathways effectively
The challenge?
