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Nutr Rev (2012) 70(Suppl 1):S3844.10.1111/j.1753-4887.2012.00493.x 175 HartstraAVBouterKEBackhedFNieuwdorpM
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This reciprocal crosstalk between adipocytes, immune cells, and stromal components establishes a feed-forward loop of inflammation, adipose tissue dysfunction, and insulin resistance, positioning the adipose immune microenvironment as a critical axis in the pathogenesis of T2DM and a promising therapeutic target
