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On the other hand, insulin promotes pancreatic cancer initiation and sustains PDA development by eliciting tumorigenic inflammation, regulating lipid and glucose metabolic reprogram, overcoming apoptosis through the crosstalk with IGF-1, stimulating cancer metastasis, and activating tumor microenvironment formation (inflammation, fibrosis, and angiogenesis)
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enm-2024-2164-Supplemental-Fig-S3.pdf Article information CONFLICTS OF INTEREST No potential conflict of interest relevant to this article was reported
Do the benefits outweigh the risks, or does safe use require heightened awareness, monitoring, or restriction
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