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Interestingly, a comparison of acute metabolic responses with the chronic profiles at the end of the treatment period strongly suggests that while the different GLP1-based agonists may share primary mechanisms of action, the durability and efficiency of their long-term metabolic responses differ, with clear superiority for the GLP1/E compound

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Clinical interest has focused on GLP-1 as GIP is no longer active in Type 2 diabetes

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Insulin is physiologically constantly secreted to a small extent during the fasting state in order to enhance glucose uptake by the peripheral tissues