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ACSL4 deletion or treatment with liproxstatin-1, an inhibitor of ferroptosis, significantly reduced 4-hydroxynonenal (4-HNE) levels, while the level of 4-HNE in esophageal cancer tissues after radiotherapy was significantly associated with better clinical prognosis of cancer patients (257)

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In addition, GLP-1R can be continuously activated to activate cAMP and P38 (94) and inhibit the conduction of the AKT and ERK1/2 signaling pathways (14, 16), ultimately inhibiting the proliferation of transplanted tumors in vivo and inducing the apoptosis of human pancreatic cancer cells in vitro ( 14)

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