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Despite these setbacks, encouraging animal data warrant future efforts for the development of new drugs capable to activate synergistically both GPR40 and GPR120 and mediate, through GLP-1 and other intestinal, pancreatic and cerebral peptides, better treatments for multifactorial chronic metabolic diseases

Laboratory research has revealed something remarkable

2021;11(2):e12432

This dose escalation strategy helps minimise gastrointestinal side effects whilst allowing the body to adapt to the medication's effects

InvivoGens cell-based metabolic assays capture receptor activity and downstream responses, providing functional insight essential for identifying bioactive compounds for therapeutic development

In the acetic-acid-induced colitis model, dulaglutide enhanced intestinal barrier function, upregulating colonic Trefoil Factor (TFF)-3 expression, as measured by enzyme-linked immunosorbent assay (ELISA) [21]