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Herein, we successfully switched a patient with acromegaly from a dipeptidyl peptidase-4 (DPP-4) inhibitor to a glucagon-like peptide-1 (GLP-1) analog due to pasireotide-induced deterioration of glycemic control, and we examined the underlying mechanism for glycemic control

AMP-activated protein kinase (AMPK) is a key enzyme broadly involved in regulating cellular metabolism, often called an energy sensor

These findings highlight the importance of targeted therapeutic strategies based on HF phenotypes

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