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In conclusion, the current study probed the function and mechanism of semaglutide in improving PCOS in vivo experiments, and found that it can be achieved via regulating the PI3K/AKT/mTOR signaling pathway to reduce oxidative stress and autophagy (Figure 9)

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Furthermore, the reduction of GAL3 in these mice reduced neuroinflammation-associated anxiety levels (Stajic et al

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