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More specifically, it has been reported that following ischemia, oxidative stress, or acidosis injury in rat caECs, CMs, and aortic SMCs, sAC-dependent apoptosis is triggered by translocation of sAC from the cytosol to mitochondria with submitochondrial PKA activation, Bax phosphorylation, and cytochrome c release (4548)

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doi:10.4049/jimmunol.1401149 57

Risk of severe COVID-1352 19 disease with ACE inhibitors and angiotensin receptor blockers: cohort study 1353 including 8.3 million people

a , e , i , Venn-diagram with number of differentially expressed genes (DEGs
