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They work through entirely different mechanisms and have very different risk profiles

Centrally, GLP-1, primarily produced by neurons in the nucleus of the solitary tract (NTS), modulates appetite and body weight through widespread neural projections (19,20)

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It becomes maddeningyou reduce your weight and youre feeling great about it, but the more you reduce your weight, the further away you get from your weight set point, which increases these signals, driving their behavior to go back and eat

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This shift creates an opportunity to build eating habits aligned with your health goals, rather than fighting constant hunger signals