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First, human amyloid- peptide (A 42 ), a toxic cleavage product of the amyloid precursor protein associated with Alzheimers disease 3, 28 was examined in five glutamatergic tail neurons using the eat-4 promoter (P eat-4 ) (Supplementary Figure S1a)

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Changes in neurogenesis are followed by decreased expression of Notch1, NICD, and Hes5 in the context of the pathogenesis of PD, suggesting that accumulation ofsynuclein may impede nasopharyngeal carcinoma (NPC) survival by impairing the Notch signaling system (Zhang et al., 2020)

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