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SARS-CoV-2-mediated dysregulation of metabolism and autophagy uncovers host-targeting antivirals

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Metabolites such as flagellin, lipopolysaccharide, peptidoglycan, and lipoteichoic acid can disrupt the tight junctions between intestinal epithelial cells and trigger inflammation by activating TLR4 and TLR2 signaling [145]

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