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Interestingly, experimental infection studies in rodent models of human gastric disease have confirmed that long-term H

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Mechanistically, TNF- acts as a central mediator by binding to its receptor tumor necrosis factor receptor 1, subsequently triggering the activation of NF-B, mitogen-activated protein kinases (MAPKs) and c-Jun N-terminal kinase (JNK) pathways (53)

[DOI] [PMC free article] [PubMed] [Google Scholar] Garvey LJ, Pavese N, Politis M, Ramlackhansingh A, Brooks DJ, Taylor-Robinson SD, Winston A (2014) Increased microglia activation in neurologically asymptomatic HIV-infected patients receiving effective ART

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The role of aldose reductase in polyol pathway: an emerging pharmacological target in diabetic complications and associated morbidities