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Furthermore, single-cell RNA sequencing-based comparisons of OPCs obtained from young and old mice have revealed mitochondrial dysfunction and a greater activity of the inflammasome and pathways associated with nutrient signaling in aged OPCs (Neumann et al., 2019)

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The body interprets sustained amylin signaling as a signal to stop eating, and sometimes that signal overshoots into actual nausea

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The dynamic interplay between p53 and glutamine metabolism underscores the delicate balance between tumor cell metabolism and the intricate signaling pathways that govern cellular responses to the tumor microenvironment [34]