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This review underscores the necessity of early recognition, accurate diagnosis, and tailored interventions to mitigate complications and enhance patient outcomes

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The GLP-1-induced enhancement of evoked EPSCs was abolished by a selective GLP-1 receptor antagonist, exendin 939, as well as by the extracellular application of a specific protein kinase A (PKA) inhibitor, KT5720

Furthermore, the SELECT results support the broader case for a weight-centric approach to CV risk management and a change in treatment guidelines

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