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GLP-1 and NMDA receptors are expressed throughout the mammalian brain and, although reports indicate that GLP-1 analogues engage central appetite circuits through distinct subpopulations of neurons (most notably in the brainstem and the hypothalamus) 36 , feedforward neural pathways modulated by inhibition of NMDA receptors specifically in GLP-1-receptor-positive neurons may differ from those engaged by exclusive activation of GLP-1 receptors

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The pulse radiolysis studies demonstrated that DSePA-mediated radioprotection of in vitro models was due to its ability to directly scavenge the radiolytically generated hydroxyl and peroxyl radicals [82, 83, 85,86,87]

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Peroxyl radicals are relatively long-lived with diffusion ability